population response to the coronavirus (children vs. adult vs. elderly) - how would a clinical trial be affected if enrolling adults vs,how the generalizability of the findings be applicable?

please I need answer in 6 lines with reference

Compare and contrast the functions and metabolism of VLDL and chylomicrons

1. Identify and describe the function of major organelles and structures in eukaryotic cells

2. Identify and describe the function of major structures in bacterial cells

1. Explain why bacterial behaviors such as biofilm formation, quorum sensing, and endospore formation can promote disease

2. Explain multiple ways in which causing disease to humans can benefit microorganisms

Medical Report: Nephrology Consultation

HISTORY OF PRESENT ILLNESS: This 57 year old white male was admitted to the hospital yesterday with a history of progressive lethargy, weakness, dysphagia, constipation, and generalized malaise. These symptoms have been present for the last 3 to 4 days.

During his last hospitalization on January 20, 2017, preoperative investigation revealed a BUN of 32 and a creatinine of 2.8, and there was no documentation of any BUN or creatinine at the time of discharge. He has a normal IVP.

It was noticed that he has a urinary tract infection with E. coli at that time, and hence he was discharged with Bactrim.

PHYSICAL EXAMINATION: Revealed a 57 year old man, a little lethargic, well oriented. His BP was 136/74 mmHg. Tongue pink and a little dry. Neck: carotid pulsations normal. Skin: decreased in turgor at present. Heart sounds normal. No gallop. Lungs with normal breath sounds. Abdomen is full, with operative scar in the right hypochondrium, with ascites present. Extremities: no peripheral edema. Well perfused. Peripheral pulsations normal.

LABORATORY: Blood chemistry on 7/23/17: Na 134. K 4.7. CO2 80. Cl 100. BUN 128. Creatinine 10.0. Random blood glucose 117. Blood chemistry on 9/20/16: Na 139. K 4.4. CO2 18. Cl 107. BUN 138. Creatinine 7.6. Fasting blood glucose 110. Ca 8.4. P 5.5. Uric acid 19.7. Total protein 5.8. Albumin 2.4. Mg 3.4. Alkaline phosphatase 41.2. SGPT 59. SGOT 62. CBC: WBC 6.2 with Hgb 13.3. Hct 38.5. platelets 246.000. Urinalysis has shown specific gravity of 1.012. No protein or hemoglobin present. WBC 2-3, RBC 0-1; bacterial cells +1.

IMPRESSION: The patient has chronic renal failure of several years' duration with compromised renal function. His postoperative course was uneventful except for complaint of slight reduction of urinary output. There has been no documentation of renal function at the time of discharge. The patient was given Bactrim, following which the patient developed symptoms of uremia, and on investigation, the patient's renal function has markedly deteriorated in the course of 5 days. It is my presumption that the patient's chronic renal failure, which was in a delicate balance, has further deteriorated with Bactrim. The Bactrim has been discontinued for 24 hours now. There has been improvement in the creatinine level, from 10 to 7.6 mg%. His BUN is still high, and it appears that the patient is still in hypovolemia and needs further hydration.

In addition, the patient has pyelonephritis of several years' duration, and analysis of the urine yesterday does not reveal any evidence of persistence of the infection. The patient might also have renal function impairment secondary to chronic pyelonephritis.

Review of the record and the patient reveals no evidence of acute ischemic renal failure. His serum magnesium is high, and the patient received some Milk of Magnesia at home. If the patient does not show any further improvement in the next 24 hours, suggest obtaining a renal scan with blood flow studies.

1) Were the results of the preoperative BUN and creatinine studies abnormal? How can you tell?

2) Which organ function is evaluated with those two laboratory tests? How do these tests work?

3) Describe an IVP procedure.

4) Why has the patient's skin turgor decreased? How can you tell?

1.Identify motility structures of protists (protozoa) that are present on a cell

2. Classify fungal cell forms as yeast, hyphal, dimorphic, or spore-producing structures

A 29-year-old female develops sepsis and, as a consequence, she experiences profound vasodilation.

a) What effect does vasodilation have on the afterload? Explain why.

b) What effect does vasodilation have on blood pressure? Explain why. How will her body try to bring her blood pressure back to homeostasis?

Be detailed in your explanation

APA FORMAT PLEASE.

Chief Complaint: 74-year-old woman with shortness of breath and swelling. History: Martha Wilmington, a 74-year-old woman with a history of rheumatic fever while in her twenties, presented to her physician with complaints of increasing shortness of breath ("dyspnea") upon exertion. She also noted that the typical swelling she's had in her ankles for years has started to get worse over the past two months, making it especially difficult to get her shoes on toward the end of the day. In the past week, she's had a decreased appetite, some nausea and vomiting, and tenderness in the right upper quadrant of the abdomen. On physical examination, Martha's jugular veins were noticeably distended. Auscultation of the heart revealed a low-pitched, rumbling systolic murmur, heard best over the left upper sternal border. In addition, she had an extra, "S3" heart sound. Top of Form

5. You examine Martha's abdomen and find that she has an enlarged liver ("hepatomegaly") and a moderate degree of ascites (water in the peritoneal cavity). Explain these findings. 6. Examination of her ankles reveals significant "pitting edema." Explain this finding. 7. She is advised to wear support stockings. Why would this help her? 8. Which term more accurately describes the stress placed upon Martha's heart -- increased pre-load or increased afterload? 9. What is the general term describing Martha's condition? 10. How might Martha's body compensate for the above condition? 11. Martha is started on a medication called digoxin. Why was she given this medication, and how does it work? 12. Two weeks after starting digoxin, Martha returns to the physician's office for a follow-up visit. On physical examination, she still has significant hepatomegaly and pitting edema, and is significantly hypertensive (i.e. she has high blood pressure). Her physician prescribes a diuretic called furosemide (or "Lasix"). Why was she given this

question about anatomy true and false questions answers

1. Ventricles have thicker, more muscular walls than atria.

True

False

2.

Conduction myofibers comprise large powerful muscles such as the biceps brachii.

True

False

3.

The sound of a heartbeat is caused by elastic recoil of large arteries.

True

False

4.

The pulse is the result of the valves closing.

True

False

5.

A surfactant is a chemical that allows water to spread farther over a surface.

True

False

6.

The myenteric plexus innervates two layers of the muscularis.

True

False

7. Brunner’s glands help neutralize stomach acid when it enters the small intestine.

True

False

8.

The falciform ligament partitions the liver into right and left lobes.

True

False

9.

The superior pharyngeal constrictors are all smooth muscle tissue.

True

False

10.

The inferior pharyngeal constrictors are mostly skeletal muscle tissue.

True

False

11.

Kuppfer cells phagocytize stuff in the liver.

True

False

12.

Fetal hearts shunt the blood from the right atrium directly to the left atrium.

True

False

13.

The pH of the duodenum is slightly acidic.

Group of answer choices

True

False

14.

The esophagus is lined with non-keratinized stratified squamous epithelium.

Group of answer choices

True

False

15. The greater omentum attaches to the greater curvature of the stomach.

True

False

16.

The liver produces bile fluid from old worn out red blood cells.

True

False

17

The vermiform appendix may act as a refuge for beneficial intestinal flora.

True

False

18.

Blood flows from the liver back to the heart through the hepatic portal vein.

True

False

Connect the pathways of β–oxidation and ketogenesis. Why is one dependent on the other

Describe the cellular mechanism in 160 words of : Binding of insulin to its receptor and the activated intracellular transduction pathways .

A key feature of kidney function involves autoregulation to maintain a nearly constant filtration fraction over a wide range of arterial pressures. Discuss the role of the macula densa and the juxtaglomerular cells that allows this type of homeostatic control to occur

discuss how altered cellular communication and chronic inflammation work in the ageing process

Which of the following would be good evidence that the negative resting potential of a cell is due to Cl- diffusing in ? (choose all that are correct)

- Anoxia causes an immediate large change in the membrane potential

- When Cl- channel blockers are applied, the membrane potential goes toward zero

- When Cl- concentrations outside the cell are raised, the membrane potential becomes more positive

-When Cl- concentrations are raised outside the cell, the membrane potential becomes more negative

a.Explain the functional roles of kidney in the maintenance of water balance in the body.

b.Illustrate the damages of blood capillaries during the disease process of diabetes.