Describe the process of ecological succession from a pioneer community to a climax community. include a discussion of species diversity and interactions accumulation of biomass and energy flow.

define epigenetics and give some examples of what and how it can alter "genetic" information/control

Question 1. Use the disease transmission model as a basis for responding to the three following multipart questions. Your answers are expected to be qualitative or semi-quantitative (e.g. more or less, higher or lower, faster or slower) only. (B) Social distancing and wearing face masks are proving to be highly effective in slowing the spread of the Coronavirus. If you were using the disease transmission model to simulate how these social behaviors slow the spread of the disease, which one factor (S, I, m, or b) in the model would you change? How would you change this particular factor (increase or decrease) to simulate slowing the spread of the disease and why? Explain.

Explain the overall FUNCTION of the extracellular matrix protein families (as a whole), such as the collagens,

fibronectins, laminins, and integrins.

Please explain it as a short answer

A cause of macrocytic anemia is:

A. Iron deficiency

B. Antibodies against intrinsic factor

C. An enzyme deficiency

D. Inheritance of abnormal hemoglobin structure

       E. None of the above

2. In sickle cell disease, vassoocclusive crisis is the result of:

        A. Sequestration of large number of RBCs in the Spleen

        B. Damage to the platelets due to antibodies

        C. plugging of small blood vessels by sickle RBCs

        D. Treatment Drugs side effects

3. Localized out pouching of heart chamber is a /an:

         A. Thrombus

         B. Embolus

         C. Aneurysm

         D. Thromboembolism

4. Which of the following is not a risk factor in development of  atherosclerosis?

   A. Diabetes

   B. Smoking

   C. Hypertension

   D. Anemia

5. The current anabolic steroids used by some gym rats are LEAST LIKELY to give them:

1. bigger muscles
2. higher LDL
3. Lower HDL
4. Testicular enlargement

Gozilla (western lowland Gorilla)

1. Social Organization (How is this species socially organized? Use term like nuclear fission/fusion, uni-male.multimale

2. Mating Strategy (monogamous, poly gamous)

3. Intermembral index

4. 3 unique facts

5. Three Unique Facts

1. Suborder and infraorder of Kikuyu Colobus monkey

2. Social Organization (How is this species socially organized? Use term like nuclear fission/fusion, uni-male.multimale

3. Mating Strategy (monogamous, poly gamous)

4. Intermembral index of Kikuyu Colobus monkey

5. Three Unique Facts

⦁   Discuss the role of calcitonin in maintaining normal blood calcium concentration.

Please explain with details!!! THANK YOU!

3.

1. Consider the following normal sequence:

Gly-leu-arg-gln-cys-ile-phe

And the following mutant proteins. What is the nature of each mutation? Describe what happens in each case.

1. Gly-leu-arg-gln                     
2. Gly-leu-arg-arg-cys-ile-phe
3. Gly-phe-lys-thr-met-gln-ile

2. Epidermolytic hyperkeratosis is an autosomal dominant condition that produces scaly skin. It can be caused by a missense mutation that substitutes a histidine (his) amino acid with an arginine (arg). Write the mRNA codons that could account for this change, and indicate if it is a transition or transversion.

c. Individuals with mutations in the GJB2 gene are deaf. The most common mutation in a population is a deletion of this gene’s 235^th nucleotide, which is a C. The normal GJB2 protein is made in the cytoplasm and then moves to the cell membrane. In patients with the most common mutant allele, the GJB2 protein remains in the cytoplasm and is quickly degraded. This mutation is likely classified as a:

1. Missense mutation
2. Nonsense mutation
3. Gain-of-function mutation
4. Loss-of-function mutation

Create a discussion question about Endocrine System and submit a thorough response to this question.

What are social, indirect, and economic costs of bacterial meningitis on a global scale? ?

Angiogenesis is an essential property of many solid tumors, and the target of several anticancer drugs. One clinically important angiogenic pathway involves vascular endothelial growth factor (VEGF) and its cognate receptors.

a) What is VEGF and how is it important in tumor growth and survival?

b). What agents target the VEGF pathway and what are their mechanisms of action?

c.) What kinds of tumors are effectively treated with agents that target the VEGF pathway?

d) What are the important toxicities of the agents that target the VEGF pathway?