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In: Biology

DCMU is marketed as an algicide and an herbicide. It works by blocking the binding site...

DCMU is marketed as an algicide and an herbicide. It works by blocking the binding site for plastoquinone on photosystem II.

  1. If DCMU blocks plastoquinone from binding to photosystem II then in what state (oxidized or reduced) will the chlorophyll a of photosystem II be stuck? Why?
  2. Based on your answer above, in what state would plastoquinone be stuck? Why?
  3. Considering where DCMU binds, would plants in nature exposed to DCMU be able to produce

ATP and NADPH through the light reactions? Why or why not?

  1. If plants exposed to DCMU cannot produce ATP and NADPH through the light reactions, how

would this affect the light-independent reactions?

  1. DCMU, as an herbicide, will eventually kill plants that are exposed to it. Why is this the case? In

answering this, relate DCMU to photosynthesis and both DCMU and photosynthesis to cellular respiration.

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