Consider the table below giving the numbers of juveniles and adults for one generation.

Calculate fitnesses (viabilities), relative fitnesses, the selection coefficient, and the heterozygous

effect. What type of selection is this? What will the allele frequencies be for the adults in the

next two generations assuming random mating? What will the frequency of the A1 allele be at

equilibrium?

Genotype: A1A1 A1A2 A2A2

Juveniles: 795 267 138

Adults: 596 242 83

Question: 1. Neatly, draw two cross sections showing differences in the arrangements of the vascular tissue... 1. Neatly, draw two cross sections showing differences in the arrangements of the vascular tissue found in a dicot herbaceous stem and a six year old dicot woody stem. Identify the following tissues if they are present: ground tissue (soft support), vascular tissue (both primary and secondary conducting tissues), wood, bark, seasonal growth rings, pith rays. (be complete)

2. Color code and explain the location of the living functional vascular tissue and location and function of the different cambiums present in both cross sections.

3. Explain how these growth rings are formed during the growth of a tree. Can the drawings be in detail please!

Why might a small deletion be viable if heterozygous?

How does a small duplication result in a phenotype when heterozygous?

Larger deletions are typically not viable even as heterozygotes. Why? And what genetic term is associated with this phenomenon?

The ubiquitin protein from the malaria parasite was treated with chymotrypsin, and the resulting fragments were sequenced. A second sample of the polypeptide was treated with trypsin, and the fragments were sequenced. What is the sequence of the polypeptide?

Chymotrypsin fragments:

AGKQLEDGRTLSDY

IPPDQQRLIF

VKTLTGKTITLDVEPSDTEIN-VKAKIQDKEGI

NIQKESTLHLVLRLRGGMQIF

Trypsin Fragments:

LR

AK

EGI

IQDK

LIFAGK

QLEDGR

TLTGK

IPPDQQR

GGMQIFVK

TLSDYNIQK

ESTLHLVLR

What is the difference between a woman with 45, X and one with 46, XX given that in all of the cells of a 46, XX individual one X chromosome is inactivated anyway?

Girls with Turner's syndrome are a little shorter than average, have a thicker neck, usually can't have children and can be socially inept - they butt into conversations, misread body language, are insensitive to others' feelings… In essence they act a lot like boys. Why might this be? (Hint: where do boys get their X chromosome?)

Using the parental Meiotic Nondisjunction rates from the class powerpoint make an argument about the % of Turner’s individuals that are socially inept versus socially competent.

8. Imagine a lake receives acid rain for an extended period of time. The original pH of the lake was 6, but it has now dropped to pH 3. How many times more acidic has this lake become? What negative impacts can this amount of acidification have on the environment?

What is responsible for the transition from the occasional or initial drug taking to habitual drug taking, and why does this not occur in everyone who has used addictive drugs? What is the

difference between the positive incentive value of a drug and the hedonic value of a drug?

Cystic Fibrosis is a disease caused by a defect in a Chloride ion transport channel protein. The channel performs a critical function in the lungs; thus, disease sufferers have trouble clearing mucous from their lungs, leaving them susceptible to opportunistic infections.

Discuss how this protein may function (normally) in the lung & hypothesize on possible treatment schemes.

What can we learn from the story of Titus about each of the following: (a) the importance of females in gorilla troop social structure (10 points) ; (b) how males compete for dominance - physically (5 pts); sexually (5 pints); socially/politically (5 points); genetically (5 points).

In Drosophila melanogaster, black body (b) is recessive to gray body (b+), purple eyes (pr) are recessive to red eyes (pr+), and vestigial wings (vg) are recessive to normal wings (vg+). These three genes are linked with pr located between b and vg at a genetic distance of 6 cM from b and 13 cM from vg. Previous experiments have shown that the interference associated with these three genes is 0.5. A fly with a black body, purple eyes and vestigial wings is crossed with a fly with a gray body, red eyes and normal wings. All of their F1 progeny is wild-type. The F1 females are mated with male flies that have black bodies, purple eyes and vestigial wings. Ten thousand progeny are produced from this testcross. How many of the testcross progeny are expected to have purple eyes but be wild-type for body color and wing type?

A. 19.5 B. 39 C. 78 D. 280.5 E. 561

Describe the 4 levels of structure of a protein and describe how the R groups influence these structures.

how does dna work

I have sequenced roughly 24 genomes from two different continents (Africa and South America), specifically for the envelope gene of yellow fever virus. I used software to generate the dN and dS ratio for each continent

Africa: (dS= 0.2065, dN= 0.0031.) dN/dS ratio= 0.0150

South America: (dS=0.5541, dN= 0.0112.) dN/dS ratio= 0.0202

Im unsure how to interpret these ratio's, and how the difference in the two suggests any differences in the type of selection for the enveloped protein in each continent.

What is the basic constraint in recombination dictated by the 12/23 rule? Why does V_kjoin to J_konly, and V_lto J_l only, and V_H to D_H to J_H only?