As mentioned in class, some inhibitory postsynaptic potentials (IPSPs) are the result of Cl - entering...

As mentioned in class, some inhibitory postsynaptic potentials (IPSPs) are the result of Cl ^- entering the cell through a chloride channel. Other IPSPs are caused by the opening of K ⁺ channels.

How could movement of the potassium ion result in hyperpolarization of the postsynaptic cell?

I get that there is movement; I need to know how or more-so why it results in hyperpolarization .

Expert Solution

Sydney Mullin answered 1 year ago

Types of postsynaptic potentials mastering biology

Types of postsynaptic potentials The binding of neurotransmitter to ligand-gated ion channels in the postsynaptic membrane causes these channels to open. As soon as the neurotransmitter is removed from the synaptic cleft, the ligand-gated ion channels close. In the brief time these channels are open, ions are able to diffuse across the postsynaptic membrane down their electrochemical gradient. The result is a postsynaptic potential, a brief change in the membrane potential of the dendrites and cell body of the postsynaptic...

If only inhibitory postsynaptic potential occurs without excitatory postsynaptic potential at all, what happens to spike...

If only inhibitory postsynaptic potential occurs without excitatory postsynaptic potential at all, what happens to spike timing dependent plasticity? in brain

Draw graphs showing the effects on action potentials in a postsynaptic cell of effective doses for...

Draw graphs showing the effects on action potentials in a postsynaptic cell of effective doses for each of the listed toxins. Assume that the cell is normally brought to threshold by the stimuli applied to its inputs, so that any abnormality is due to the toxin. 1. curare (prevents receptor from binding neurotransmitter) 2. botulinum toxin (prevents neurotransmitter release)

Compare and contrast excitatory and inhibitory post-synaptic potentials. In your answer, address the following: How are...

Compare and contrast excitatory and inhibitory post-synaptic potentials. In your answer, address the following: How are they generated? What is the outcome? How might their size or strength be increased or decreased? Why might you want to modify synaptic strength? Provide a mechanism.

Describe what causes excitatory and inhibitory post-synaptic potentials, and how each affect a post-synaptic neuron.

Use standard reduction potentials to calculate ΔG° for the reaction: ClO4- + 6 Cl- + 6...

Use standard reduction potentials to calculate ΔG° for the reaction: ClO4- + 6 Cl- + 6 H3O+ →ClO- + 3 Cl2 + 9 H2O E°ClO4-/ClO-, H3O+ = +1.36, E°Cl2/Cl- = +1.35827

A college professor claims that the entering class this year appears to be smarter than entering...

A college professor claims that the entering class this year appears to be smarter than entering classes from previous years. He tests a random sample of 14 of this year's entering students and finds that their mean IQ score is 116, with standard deviation of 14. The college records indicate that the mean IQ score for entering students from previous years is 111. If we assume that the IQ scores of this year's entering class are normally distributed, is there...

Survey some of the mechanisms by which drugs can alter presynaptic and postsynaptic processes.

Using the reflex pathways explain why inhibition of inhibitory interneurons might result in uncontrollable muscle spasms?

In a large midwestern university (the class of entering freshmen is 6000 or more students), an...

In a large midwestern university (the class of entering freshmen is 6000 or more students), an SRS of 100 entering freshmen in 1999 found that 20 finished in the bottom third of their high school class. Admission standards at the university were tightened in 2000. In 2001, an SRS of 100 entering freshmen found that 10 finished in the bottom third of their high school class. Let p1 and p2 be the proportion of all entering freshmen in 1999 and 2001, respectively, who...

Question