In: Nursing
CLINICAL SCENARIO
NURSING HEALTH HISTORY
A. Patient’s Profile
Name: JFK
Birthday: August 23, 1982
Age: 38 years old
Sex: Male
Nationality:Filipino
Religion:Roman Catholic
Marital Status:Married
Address: Pampanga
Date of Admission: September 02, 2020
Time of Admission: 2:30PM
Chief Complaint: productive cough with fever with slight difficulty of breathing
Final Diagnosis: Moderate Risk Community Acquired Pneumonia (CAP III)
History of Present Illness :
Patient came to the hospital complaining of productive cough, fever and slight difficulty of breathing. Client is having persistent productive cough with greenish phlegm and has had fever with 39.3 celcius for temperature when admitted. The client is ambulatory, coherent and v/s results showed an elevated RR of 37 cpm, pulse rate of 104. On DAT was prescribed, has a standing order of TSB for fever. WBC count is within range, CXR results showing consolidation and sputum culture and sensitivity shows S. pneumoniae with a medical diagnosis of CAP III.
Past Medical History :
Cough and fever has been noted to have onset 4 days prior to admission. Client has history of pneumonia and was admitted to the hospital when he was in high school. Client has suffered asthmatic attack when he was 3 years old and was admitted to the hospital and was given Ventolin for treatment but has no record of any onset after that.Immunizations were completed when he was one year old.Latest medicines prescribed are Cefuroxime, Albuterol, Montelukast, and Naproxen. The patient never undergone any type of surgery. He has no known allergy to food and medication.Family History(+) Hypertension-father(+) Diabetes Mellitus-father(-) Cancer(+) PTB-mother
Personal and Social History : Patient is a tricycle driver and a very joker person, he mingles with his co-tricycle drivers, friends, and neighbors. Patient is non-smoker, non-drinker and no history of taking illicit drugs. He prefers to eat rice, fish and vegetables, but sometimes eat in a sari-sari store. He enjoys talking to commuters and taking care of his children when his day-off, his leisure time is watching TV with his family. If the patient has free time from driving, he likes going to mall with hisfamily every Sunday after church. He is a sweet and loving husband to his wife and children.
Admission Order Medication: Cefuroxime (Zinacef) 750 mg every 8 hours TIV Levofloxacin (Levox) 500 mg 1 tab OD PO Paracematol1 amp TIV for temp of equal or greater than 38.6 and paracetamol 500mg tab PO for temp of 38.5 below Berodual nebulization (10gtts in 3 ml NSS) every 6 hour
Therapeutics: IVF PNSS 1L to run for 8 hours at 31-21 gtts per min at left arm
Bedside O2 of 3L/min via nasal cannula
Nebulization followed by CPT
Bedside Care: Vital signs every shift and watch out for any signs of dyspnea progression, bed rest, I and O monitoring, suction secretions when necessary, provide comfort, morning care done, side rails up for safety, assess every now and then and relayed any abnormal symptoms and complications
COURSE TASKS:
1. Make an Anatomy and Physiology of Community Acquired Pneumonia III.
2.Conceptualize the pathophysiological alterations distinct to the case.
✓Establish the pathophysiological triad of Host –Agent –Environment specific to the case.
✓Trace the pathophysiological changes and highlight problems that are experienced by the client.
✓Connect the pertinent nursing care and medical –surgical management to the various signs and symptoms presented by the client.
1. Pneumonia is a type of lung infection. It can cause breathing
problems and other symptoms. In community-acquired pneumonia (CAP),
you get infected in a community setting. It doesn’t happen in a
hospital, nursing home, or other healthcare center.
Community-acquired pneumonia is defined as pneumonia that is
acquired outside the hospital.
Many germs can grow inside your body and cause disease. Specific
types of germs can cause lung infection and pneumonia when they
invade. These germs can cause your respiratory system to work
poorly. That can cause shortness of breath. If your body can’t get
enough oxygen to survive, pneumonia may lead to death.
These germs can spread from person to person. When someone infected
with one of these germs sneezes or coughs, you might breathe the
germs into your lungs. If your immune system doesn’t kill the
invaders, the germs might grow and cause pneumonia.
CAP can result from infection with many types of germs. These
include bacteria, viruses, fungi, or parasites. Symptoms from
pneumonia can range from mild to severe. Certain types of germs are
more likely to lead to serious infection.
CAP is very common, especially during the winter months. It is more
common in older adults. But it can affect people of any age. It can
be very serious. That’s often the case in older adults or people
with other health problems.
2. Pathophysiology with agent host environments involvement
Pneumonia indicates an inflammatory process of the lung parenchyma
caused by a microbial agent. The most common pathway for the
microbial agent to reach the alveoli is by microaspiration of
oropharyngeal secretions. Once microorganisms reach the alveolar
space, they cause pneumonia by overcoming the last defense
mechanism of the lung, the alveolar macrophage. Most of the time,
the alveolar macrophage phagocytizes and kills the microorganisms
that reach the alveolar space. This explains why even though the
arrival of microorganisms into the alveolar space is a
not-infrequent occurrence, the presence of clinical pneumonia is
infrequent.
If the alveolar macrophage is unable to control the growth of the
microorganisms, then, as a final protective defense mechanism, the
lungs develop a local inflammatory response. This local
inflammatory response is characterized by movement of white blood
cells, lymphocytes and monocytes from the capillaries into the
alveolar space.
The recruitment of phagocytic cells to the alveolar space is
primarily mediated by tumor necrosis factor (TNF) and interleukin-l
(IL-I) produced by the alveolar macrophages. In addition to TNF and
IL-I, other important locally produced cytokines include IL-6,
IL-10, IL-12, monocyte chemotaxin protein-l and granulocyte
colony-stimulating factor (1). Once these cytokines reach the
systemic circulation, they also produce a systemic inflammatory
response. The local and systemic inflammatory response is
responsible for the majority of the signs, symptoms ( cough, sputum
production, shortness of breath, pleuritic chest pain, fever,
chills. Tachycardia, tachypnea, rales, and signs of consolidation
on physical examination) , and laboratory abnormalities that
characterize the community-acquired pneumonia a (CAP)
syndrome.
Nursing care according symptoms:-
▪Deep breathing exercises facilitates maximum expansion of the
lungs and smaller airways, and improves the productivity of
cough.
▪Coughing is a reflex and a natural self-cleaning mechanism that
assists the cilia to maintain patent airways. It is the most
helpful way to remove most secretions.
▪Splinting reduces chest discomfort and an upright position favors
deeper and more forceful cough effort making it more
effective.
▪Promoting good fluid intake through the use of intravenous fluids
will help promote release of secretions.
▪Humidified oxygen therapy could also be helpful.
Medical treatment:-
Empiric therapy with macrolides, fluoroquinolones, or doxycycline.
Patients who are hospitalized should be switched from parenteral
antibiotics to oral antibiotics after their symptoms improve, they
are afebrile, and they are able to tolerate oral medications.