Given the criticism Eleanor has received, what specific type of grief is she most likely experiencing? How might it be contributing to Eleanor’s prolonged pain, and what can you do to help her?

What other types of grief do you know?

Eleanor feels guilty for grieving like this for a patient and worries that she is experiencing depression. Based on what you know, how would you answer? What is the difference between normal grief and major depressive disorder?

This story mentions Eleanor’s participation in celebrating Maria’s life on Facebook. What is your impression of this as a positive or negative contribution—given what you’ve learned from the text?

1. Explain In your own words how the holistic nursing journey affects they way nurses care for themselves
2. In your own words explain the evolving role of a holistic nurse

A 2 year old child is in for her well visit. She is up to date with all vaccinations. Born full term, 7 pounds and 5 ounces. No allergies and not on any medications. Breast fed for six months. Mother is anxious because she is not far along in language development as her brother was at this age. She cries when getting a hair cut and is not consolable. Only eats noodles and toast. Is not a good sleeper.

1. The mothers asks, "are these things related?" How do you reply?

2. What are some online tools to screen for assessing language delays?

3. What should be included in the health history?

4. What should the focused physical examination include?

Case Study #11

History and Physical Examination:

A 24-year-old female with a history of AIDS presents for evaluation of left-sided weakness. She has also experienced headaches and seizures and others have observed an alteration in her mental status. Her medical history is notable only for an episode of Pneumocystis jiroveci pneumonia, primary syphilis treated with penicillin 5 years ago and occasional thrush. She takes zidovudine and monthly-aerosolized pentamidine for Pneumocystis prophylaxis. An urgent CT scan of the head shows two 1 cm lesions in the right basal ganglia with enhancement seen with intravenous contrast media. Laboratory data: CD4 cell count 50 cells/μL (reference 600 – 1500/μL) RPR positive 1:2 dilution Toxoplasmosis gondii IgG positive Toxoplasmosis gondii IgM negative

Questions:

1.What is the most likely cause of the lesions in the brain?

2. Is this a newly acquired infection? Why or why not?

3. What is the difficulty in using serologic techniques to diagnose infectious disease in this type of patient?

4. Should pregnant women be tested for this microorganism and why?

5. What prenatal serology is currently recommended relative to Toxoplasma past or current infection?

6. What test is performed on the newborn to aid in the diagnosis of congenital toxoplasmosis? 7. What 3 ways can a person become infected?

Assessment Background:

You are the nurse manager at a skilled nursing facility. There has been tremendous nurse burnout and turnover at your facility. Next Tuesday, the CEO and CFO of your organization are coming to assess the situation. You must explain to these stakeholders the current trends for nursing leadership related to nursing recruitment, retention, and engagement in the workplace. The goal of your presentation is to help influence your stakeholders to increase funding and make allowances for you to create positive change for a healthier workplace.

Assessment Instructions:

1. Create a 5–10-slide PowerPoint® presentation, excluding the title and reference slide(s), for the CEO and CFO that includes the following information:
   1. Analyze reasons for nurse burnout and the high turnover rate;
   2. Evaluate the best methods for the nurse manager to increase recruitment and retention;
   3. Correlate the relationship between staffing, productivity, and quality leadership for employee engagement.

Which factors affect cardiac output? What types of changes in cardiac output can be made to try and treat hypertension and congestive heart failure? Many classes of drugs affect cardiac output, which one is in JNC 8 protocol.

Has Greg provided accurate information concerning Mrs. Angstrom’s options for advance directives? If not, what’s wrong with what he said? What options would be more appropriate to suggest to her?

Mention at least 4 facts and 4 myth about aging, and explain one of then.

Has Greg provided accurate information concerning Mrs. Angstrom’s options for advance directives? If not, what’s wrong with what he said? What options would be more appropriate to suggest to her?

Mention at least 4 facts and 4 myth about aging, and explain one of then.

HbA1c is a blood test used in the management of diabetes. Describe what this test identifies.     

Describe how HbA1c levels might help a registered nurse or diabetes educator manage a patient’s diabetes.

List 4 signs and symptoms of acute respiratory distress.

Describe four (4) nursing interventions in the direct post-angiogram period for a patient who had an angiogram via the right radial artery.   

Read the assessment background below and answer the questions that follow
Assessment Background:

You are the nurse manager at a skilled nursing facility. There has been tremendous nurse burnout and turnover at your facility. Next Tuesday, the CEO and CFO of your organization are coming to assess the situation. You must explain to these stakeholders the current trends for nursing leadership related to nursing recruitment, retention, and engagement in the workplace. The goal of your presentation is to help influence your stakeholders to increase funding and make allowances for you to create positive change for a healthier workplace.

Questions:

1. Provide a basic analysis of reasons for nurse burnout and high turnover rate.

2. Thoroughly and insightfully evaluate the best methods for the nurse manager to increase recruitment.
3. Thoroughly and insightfully evaluate the best methods for the nurse manager to increase retention.

4. Described the relationship between staffing, productivity, and quality leadership for employee engagement.

Discuss at least 3 symptoms caused by physiologic changes and consequences of hypertension. Discuss how pharmaceutical treatments are designed to alleviate those symptoms. What are the risks and benefits of these treatments? What are the potential drug interactions with these medications? What added therapy would you consider if the patient's hypertension were complicated by heart failure? What therapies would you avoid? What patient education is needed in terms of compliance and expectations of medications prescribed

By the spring of 1934, a great deal was known about poliomyelitis. The mode of transmission was known to be person-to-person. The two-phase process of the disease was well understood, and mild non-paralytic infections or anterior poliomyelitis as well as paralytic infections were all understood to be major means of contagion. Animals and most insects were eliminated as vectors. It was known that some victims will die in a few days. Some would have crippling paralysis, and others would recover without a sign. The polio virus had been isolated and identified from most parts of the body---most importantly, the CNS; blood; saliva; gastrointestinal tract, especially the small intestine; mesenteric lymph nodes; and nasopharynx. The damage caused by the polio virus was known to be done in the spinal cord's anterior horn of the grey matter and in the brain tissue.*

When the poliomyelitis epidemic hit Los Angeles, many horror stories from past epidemics had been deeply planted in the minds of medical and nursing professionals. It appears that the medical professionals at the time were well informed about the facts of poliomyelitis, yet most ignored them and, moreover, failed to inform the public. The Contagious Unit of the Los Angeles General Hospital was responsible for most of the activities of the epidemic, and fear of the disease seemed to dominate its efforts, in spite of evidence that much of the sickness that occurred in June of 1934 was not poliomyelitis.*

Physicians and nurses were strained, worried, and terrified of contracting the disease themselves. By June 15, 50 cases a day were being admitted to most hospitals, yet by June 29, only 1 fatal case of poliomyelitis had occurred, producing a sample of the polio virus. A second case produced another sample on July 4.*

When the Poliomyelitis Commission arrived in Los Angeles from Yale University School of Medicine, headed by Dr. Leslie T. Webster of the Rockefeller Institute of Medical Science of new York City, a public meeting was held to review the situation of the epidemic. The meeting digressed to physicians and nurses discussing their risk of getting poliomyelitis and whether they might receive disability pensions if paralyzed by the disease and were disabled in the line of duty.*

New interns in training at the Los Angeles County Hospital were deprived of teaching and proper guidance because the attending physicians were afraid of getting the disease and stayed away, consulting by phone instead of going to the hospital. Doctors who worked at the County Hospital in the communicable disease wards were not welcome on house calls because their patients viewed the hospital as a pet house.*

No one knew how much of the disease that year was really polio. Nearly all adults, especially the nurses and doctors, were afraid of getting paralytic polio. In those who got the serious form of the disease, health care providers observed much pain and weakness, but very few deaths occurred. The number of cases of paralysis was much lower than one would expect. The question was this: Could it be another virus or different strain of the virus? Dr. Webster believed that 90% of the cases were actually not poliomyelitis.*

Researchers had little success in searching for the polio virus in the nasal passages of suspected victims through nasal washings. The disease could not be produced in monkeys or lab animals. Webster believed that the problem was complex and that the infantile part of the infantile paralysis was missing because most cases were in adults. The paralysis face of the disease was also missing, as no paralysis occurred in most cases.*

Oral washings with ropy (an adhesive, stringy-type thread that was soaked in a special solution and swirled around in the throat in order to capture samples of mucous tissue) were done routinely. Ropy washes were able to gather even a few flakes of mucous and the debris in it. The ropy washes used a special solution that helped save samplings of potential polio virus evidence and preserved the evidence for months (101) days for later study. Even after such a long time, the specimen could be spun in a centrifuge and yield the virus; thus, in future outbreaks, disease investigators would not need to take an army of public health workers along to gather specimens.*

Hysteria raged on in the main populace. Not only was the general public afraid of getting the disease, but a major part of the medical and nursing profession was also participating in the fear. Yet officials were not daring enough to tell the public that the disease was not polio. It was disclosed that half of the 1,301 suspected cases were not poliomyelitis. The actual attack rate was estimated to be from 4.4% to 10.7%.*

There was no doubt that Los Angeles was visited by the epidemic of poliomyelitis in the summer of 1934, but it was a mild one. Most of the people who were sick that summer were sick either from another disease (encephalitis, meningitis, or influenza) or from a mild form of a different strain of the poliomyelitis. Patients had atypical symptoms for polio, and the observed symptoms were rheumatoidal or influenzal with striking emotional tones of fear that they might get polio. It was observed by US Public Health Service officer Dr. A. G. Gilliam, of the Los Angeles County Hospital"s personnel, "Irrespective of actual mechanisms of spread and identity of the disease, this outbreak has no parallel in the history of poliomyelitis or any other CNS infections"*

As an unfortunate outcome of this epidemic and its resulting hysteria, patients who exhibited even a slight degree of weakness were immobilized in plaster casts. This was a common practice in the 1930s, and many were subjected unnecessarily to this treatment.

Answer the following three question below

Case Questions to Respond to

1. By 1934, a great deal was known about poliomyelitis. Summarize all that was known about all facets of the epidemiology of polio.

2. How serious was the polio epidemic of 1934?. What were the social, psychological, and political implications and their effects on the epidemiology of polio surrounding this case?

3. What were the final conclusions about the polio epidemic of 1934 in Los Angeles, and what were the implications for the future?

By the spring of 1934, a great deal was known about poliomyelitis. The mode of transmission was known to be person-to-person. The two-phase process of the disease was well understood, and mild non-paralytic infections or anterior poliomyelitis as well as paralytic infections were all understood to be major means of contagion. Animals and most insects were eliminated as vectors. It was known that some victims will die in a few days. Some would have crippling paralysis, and others would recover without a sign. The polio virus had been isolated and identified from most parts of the body---most importantly, the CNS; blood; saliva; gastrointestinal tract, especially the small intestine; mesenteric lymph nodes; and nasopharynx. The damage caused by the polio virus was known to be done in the spinal cord's anterior horn of the grey matter and in the brain tissue.*

When the poliomyelitis epidemic hit Los Angeles, many horror stories from past epidemics had been deeply planted in the minds of medical and nursing professionals. It appears that the medical professionals at the time were well informed about the facts of poliomyelitis, yet most ignored them and, moreover, failed to inform the public. The Contagious Unit of the Los Angeles General Hospital was responsible for most of the activities of the epidemic, and fear of the disease seemed to dominate its efforts, in spite of evidence that much of the sickness that occurred in June of 1934 was not poliomyelitis.*

Physicians and nurses were strained, worried, and terrified of contracting the disease themselves. By June 15, 50 cases a day were being admitted to most hospitals, yet by June 29, only 1 fatal case of poliomyelitis had occurred, producing a sample of the polio virus. A second case produced another sample on July 4.*

When the Poliomyelitis Commission arrived in Los Angeles from Yale University School of Medicine, headed by Dr. Leslie T. Webster of the Rockefeller Institute of Medical Science of new York City, a public meeting was held to review the situation of the epidemic. The meeting digressed to physicians and nurses discussing their risk of getting poliomyelitis and whether they might receive disability pensions if paralyzed by the disease and were disabled in the line of duty.*

New interns in training at the Los Angeles County Hospital were deprived of teaching and proper guidance because the attending physicians were afraid of getting the disease and stayed away, consulting by phone instead of going to the hospital. Doctors who worked at the County Hospital in the communicable disease wards were not welcome on house calls because their patients viewed the hospital as a pet house.*

No one knew how much of the disease that year was really polio. Nearly all adults, especially the nurses and doctors, were afraid of getting paralytic polio. In those who got the serious form of the disease, health care providers observed much pain and weakness, but very few deaths occurred. The number of cases of paralysis was much lower than one would expect. The question was this: Could it be another virus or different strain of the virus? Dr. Webster believed that 90% of the cases were actually not poliomyelitis.*

Researchers had little success in searching for the polio virus in the nasal passages of suspected victims through nasal washings. The disease could not be produced in monkeys or lab animals. Webster believed that the problem was complex and that the infantile part of the infantile paralysis was missing because most cases were in adults. The paralysis face of the disease was also missing, as no paralysis occurred in most cases.*

Oral washings with ropy (an adhesive, stringy-type thread that was soaked in a special solution and swirled around in the throat in order to capture samples of mucous tissue) were done routinely. Ropy washes were able to gather even a few flakes of mucous and the debris in it. The ropy washes used a special solution that helped save samplings of potential polio virus evidence and preserved the evidence for months (101) days for later study. Even after such a long time, the specimen could be spun in a centrifuge and yield the virus; thus, in future outbreaks, disease investigators would not need to take an army of public health workers along to gather specimens.*

Hysteria raged on in the main populace. Not only was the general public afraid of getting the disease, but a major part of the medical and nursing profession was also participating in the fear. Yet officials were not daring enough to tell the public that the disease was not polio. It was disclosed that half of the 1,301 suspected cases were not poliomyelitis. The actual attack rate was estimated to be from 4.4% to 10.7%.*

There was no doubt that Los Angeles was visited by the epidemic of poliomyelitis in the summer of 1934, but it was a mild one. Most of the people who were sick that summer were sick either from another disease (encephalitis, meningitis, or influenza) or from a mild form of a different strain of the poliomyelitis. Patients had atypical symptoms for polio, and the observed symptoms were rheumatoidal or influenzal with striking emotional tones of fear that they might get polio. It was observed by US Public Health Service officer Dr. A. G. Gilliam, of the Los Angeles County Hospital"s personnel, "Irrespective of actual mechanisms of spread and identity of the disease, this outbreak has no parallel in the history of poliomyelitis or any other CNS infections"*

As an unfortunate outcome of this epidemic and its resulting hysteria, patients who exhibited even a slight degree of weakness were immobilized in plaster casts. This was a common practice in the 1930s, and many were subjected unnecessarily to this treatment.

Answer the following three question below

Case Questions to Respond to

1. By 1934, a great deal was known about poliomyelitis. Summarize all that was known about all facets of the epidemiology of polio.

1. Give me a possible research introduction for a research topic "How to manage Anxiety and Depression as a Nurse During this Pandemic".

2. What are the possible questions for a research topic " How to manage Anxiety and Depression as a Nurse During this Pandemic".

There are no results for Drugs of Abuse Mr. V, a 47 year-old man, was recently promoted as a director of a transportation company. A routine inspection of the books shows that a large sum of money is missing. Subsequent investigation finds that Mr. V has been spending more than $20,000 a month to buy cocaine; currently he consumes 2-3 g/d. He also drinks several beers each day and 5-8 shots of vodka in the evening. He spends weekend nights in clubs, where he often consumes 2-3 pills of ecstasy. He began using drugs at age 18; during parties he mostly smoked cannabis (5-6 joints per weekend), but also tried cocaine. This “recreational use” came to an abrupt halt when he married at age 27 and entered a professional training program that allowed him to obtain his current job, now jeopardized by his cocaine use. 1. Is Mr. V addicted, dependent, or both? Explain your reasoning. (5points) 2. What is the reason for the use of several different addictive drugs at the same time? (5points) 3. Exogenous cannabinoids (marijuana) include several pharmacologically active substances, but the main psychoactive alkaloid is called __________________________. (2points) 4. When CB1 receptors are activated by marijuana it produces several effects. List at least 3 of the effects. (3points) 5. Ecstasy is a psychoactive drug primarily used for recreational purposes. Ecstasy increases the activity of three brain chemicals. Describe the three brain chemicals that it increases. (5points)