In: Biology
what is the difference between escherichia vulneris and hafnia alvei
Both taxa Escherichia.vulneris and Hafnia alvei were naturally sensitive or sensitive and of intermediate susceptibility to aminoglycosides, acylureidopenicillins, ticarcillin, several cephalosporins, carbapenems, aztreonam, quinolones, folate pathway inhibitors, and nitrofurantoin.
They were naturally resistant to tetracycline, penicillin G, oxacillin, all macrolides except for azithromycin, lincosamides, streptogramins, glycopeptides, rifampicin, and fusidic acid.
Taxon-related differences in natural susceptibility affecting clinical assessment criteria were seen with doxycycline, minocycline, aminopenicillins, some cephalosporins, azithromycin, and fosfomycin.
E. vulneris was more susceptible than H. alvei to these agents and was naturally sensitive to all ?-lactams, azithromycin, and fosfomycin.
H. alvei was naturally resistant or of intermediate susceptibility to all tetracyclines, amoxicillin, amoxicillin-clavulanate, ampicillin-sulbactam, narrow-spectrum cephalosporins, azithromycin, and fosfomycin.
Motile malonate-negative Hafnia strains were less susceptible to some cephalosporins than nonmotile, malonate-positive hafniae.
Proline deaminase, hydroxyproline amidase, tripeptidase, chitinase, Voges-Proskauer reaction, and assimilation of histidine as well as acid production from glycerol, rhamnose, and xylose were suitable tests to separate strains of E. vulneris from those of the H. alvei complex.