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44. Explain the pathophysiology of the condition that results from too little ADH. What is it...

44. Explain the pathophysiology of the condition that results from too little ADH. What is it called?

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Expert Solution

The condition is known as diabetes insipidus.

Diabates insipidus is a medical condition in which the charastics are increased urine output and increased thurst. This condition is caused by the defficiency of the anti diuretic hormone secretion.This increased urine output volume will leads to dehydration and fall in blood pressure value.

DIABETIS INSIPIDUS is the cause of the abnormality in the functioning of diabetes or the levels of importance of antidiuretic hormone (ADH) and vasopressin, as is well known. Made in the hypothalamus and pituitary gland, ADH helps regulate body fluid.

In healthy individuals, body fluid, the pituitary gland, is drained, urinary excretion is released from its ADH, prevents body fluids from forming. To increase the water permeability of ADH blood acts through the collecting duct and proximal convoluted tubules. Of course, to open Aquaporins ADH will act as a protein, called the water collecting duct. When it will rise and the permeability, the water, and on the other hand, is not absorbed into the blood, urine and reduces the volume and for the promotion of the contemplation of the forces have their defects.

PATHOPHYSIOLOGY of DIABETES INSIPIDUS:

Fluid and electrolyte homoeostasis maintains healthy kidney urine by adjusting physiological composition and volume needs. And the final composition of the urine is determined in the last segment of the tubular, the collecting duct. The first victim was water permeability in the arginine vasopressin collecting duct (AVP). The secretion of AVP Noctiluca is regulated by a signaling network that involves complexes of osmosensors, barosensor sensors, and rollers. AVP facilitates the reabsorption of water by aquaporin (AQP), the mediated activation of vasopressin V2 (AVPR2) in the collecting duct, allowing to avoid urine concentration. Nephrogenic diabetes insipidus, in (NDI), the kidney cannot function corresponds to AVP AQP results in a decrease. Affected patients have such a consistent diuretic that large volumes of diluted urine should be avoided. The first is the form of the effect of changes in AVPR2 NDI genes and proteins, since a key and AQP2 encode, or are secondary, are forms of obstructive abnormalities of uropathy associated with the use of certain drugs of diameter, in particular lithium.


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