1. Hormone responsible for male secondary sex characteristics is:

1. LH
2. FSH
3. Testosterone
4. ADH

2. As a result of renin-angiotensin-aldosterone system:

1. Aldosterone is secreted from pituitary gland
2. Arteriolar vasoconstriction occurs
3. ADH is produced by adrenal cortex
4. Arteriolar vasodilation occurs

3. Which hormones enter cells and then decreases or increases mRNA synthesis:

1. Steroid and peptide hormones
2. Peptide hormones
3. Steroid hormones
4. Phospholipid hormones

4. Cyclic AMP is a common second messenger, which are associated with what type of hormones?

1. Peptide
2. Steroid
3. Both A & B
4. Fatty acids

5. Which of following is a granulocyte?

1. B lymphocyte
2. T lymphocyte
3. Plasma cell
4. Neutrophil

pre lab questions:

1. Define “acid” according to the Bronsted-Lowry theory of acids and bases.

2. Hypothesize what might occur to the sodium bicarbonate when it reacts in Part B of this experiment. What will you see?

3. The NaI might undergo one of two different reactions when it reacts with 18 M sulfuric acid in Part C of this experiment. Describe what happens in both of these possibilities (in words or with reaction equations)

4. Lookup the physical appearance of I₂ and Cl₂. What do they look like?

5. Hypothesize what might occur to the magnesium and copper metals when they react with HCl in Part D of this experiment.

(BioChem) Acyl-CoA dehydrogenase deficiencies are diseases related to the impaired ability to oxidize fatty acids via beta oxidation. Symptoms of these acyl-CoA dehydrogenase deficiencies include hypoketosis [low blood levels of ketone bodies], hypoglycemia [low blood glucose], and hyperammonemia [increased blood levels of ammonia] during long periods of fasting. (Please type response, thanks!)

a.) In what 2 ways is beta oxidation important during prolonged periods of fasting?

b.) Please explain why patients with acyl-coA dehydrogenase deficiencies have bouts of hypoketosis [1- 2 sentences is sufficient].

c.) Please explain why patients with acyl-coA dehydrogenase deficiencies have bouts of hypogycemia [1- 2 sentences is sufficient].

d.) Please explain why patients with acyl-coA dehydrogenase deficiencies have bouts of hyperammonemia [1- 2 sentences is sufficient].

What are nucleic acids/nucleotides? o Overall structure (what are the 3 subunits of nucleotides?) o What types of nucleic acids are there?

· How do nucleotides/bases pair up in DNA (complementary base pairs)?

· What is genome, chromosome, gene, mutation, exon, intron, and locus?

· What is gene expression? What is made during transcription? What is made during translation? What does RNA polymerase do? What does a ribosome do?

· How are DNA and RNA similar? How are they different?

· How do nucleotides/bases pair up in RNA (complementary base pairs)?

· What are mutations? Point mutations vs. frameshift mutations?

· What is biotechnology? What are the four steps of biotechnology? What do restriction enzymes do? What does Polymerase Chain Reaction (PCR) do? What is a plasmid?

· What is genetic engineering? · What is gene therapy?

· What is DNA Fingerprinting?

· How do prokaryotic cells divide? What is binary fission?

· How do somatic eukaryotic cells divide?

· How do gametes (sperm/egg cells) divide?

· What are the stages of mitosis? What happens in each stage?

· What are the stages of meiosis? What happens in each stage?

· What are the stages of interphase? What happens in each stage?

· How is DNA replicated?

· What are checkpoints? Where are they located in the cell cycle? Why are they important?

· Know the difference between sister chromatids and homologous chromosomes.

· What is cancer? What is contact inhibition?

· Know the difference between haploid and diploid.

· What is crossing over and when in the cell cycle does it occur?

· What is nondisjunction?

Why are human nerve fibers rich in sphingolipids and glycosphingolipids? What is the purpose of these biological molecules? Explain and cite your sources.

this question is from"Human resources" subject.
What different types of information are available to Canadian managers regarding incentive plans?