the peotide bond between two amino acid is a covalent bond. true or false?

in a _____ pathway, macromolecules are broken diwn into thier constituents.

the ions Na + has more ______ than _______.

__________ capitalize the teaction by lowering the ______________.

Intermediates of the citric acid cycle can participate in which of the following metabolic pathways?

Group of answer choices

gluconeogenesis

fatty acid anabolism

heme biosynthesis

amino acid anabolism

all of the above

With the aid of a simple generic diagram, identify and explain how the type(s) of chemical bonding stabilizes a secondary structure that is present in crystal structure of Yeast Hexokinase PII with the correct amino acid sequence

9. Which of the following statements about the building blocks of polymers is true? a. When looking at the complete structure of a nucleotide you can always tell whether it is considered a ribo-nucleotide or a deoxyribo-nucleotide. b. When looking at a glucose monomer (in linear form), you can always tell which polymer of glucose (a starch or cellulose) it will be incorporated into. c. When looking at a single amino acid, you can always tell whether it will be the last amino acid in a polypeptide chain. d. When looking at a nitrogenous base, you can always tell if it will be incorporated into a doublestranded nucleic acid or a single-stranded nucleic acid. e. There is more than one correct answer

QUESTION 33

You have added a drug that reduces the activity of the enzyme topoisomerase (gyrase). What will happen to the rate of activity of the helicase at the replication fork? Explain your reasoning.

Why do you think the enzyme(PPO) activity is dropping with time? Is benzoquinone a reactive species?

ppo=polyphenol oxidase

the question comes from enzymatic browning reaction.

Compare and contrast regulation of gene expression with use of an enzyme inhibitor. List one advantage and disadvantage for regulating gene expression and one advantage and one disadvantage for using inhibitor.

3. What product is formed when sulphanilamide binds to the enzyme dihydropteroate synthase rather than the natural substrate PABA? How does this result in the inhibition of bacterial growth?